"Turmeric magnificently illustrates the benefit of the great culinary traditions, in comparison to the consumption of isolated substances. When researcher in Taiwan tried treating cancerous tumors with turmeric delivered in capsules, they discovered that it was very poorly absorbed by the digestive system. In fact, when it is not mixed with pepper or ginger -- as it always has been in curry -- turmeric does not pass the intestinal barrier. Pepper increases the body's absorption of Turmeric by 2,000 percent. Indian wisdom has thus been far ahead of modern science in the discovery of natural affinities between foods.
"When I was researching information on my own cancer, I was astonished to find out that even brain tumors such as glioblastomas were more sensitive to chemotherapy when curcumin was prescribed at the same time.
"According to the Aggarwal team in Houston, turmeric's extraordinary effect seems to be due in large part to its capacity to interfere directly with the black knight of cancer we identified in chapter 4, NF-Kappa B, which protects cancer cells against the body's defense mechanisms. The entire pharmaceutical industry is looking for new, nontoxic molecules capable of fighting this mechanism of cancer promotion. It is now known that curcumin is a powerful NF-kappa B antagonist, while over two thousand years of daily use in indian cooking has proved that it is totally innocuous. Turmeric can also be eaten with soy products that replace animal proteins and provide the genistein mentioned above, which detoxifies and helps check angiogenesis. Add a cup of gree tea and imagine the powerful cocktail that, with no side effects, keeps in check three of the principal mechanisms of cancer growth."
- Servan-schreiber, D., Anticancer: A New Way of Life. Viking (2009).
- Carter, A., " "Curry Compound Fights Cncer in the Clinic," Journal of the National Cancer Institute (2008). p. djn141.
- Cheng, A.L., C. H. Hsu, J. K. Lin, et al., "Phase I Clinical Trial of Curcumin, a Chemoprotective Agent, in Patients with High-Risk or Pre-Malignant Lesions," Ancitcancer Research 21, no. 4B (2001): 2895-900
- Shoba, G., D. Joy, T. Joseph, et al., "Influence of Piperine on the Pharmacokinetics of Curcumin in Animals and Human Volunteers," Planta Medica 64, no. 4 (1998): 353-56.
- Gao, X., D. Deeb, H. Jiang, et. al., "Curcumin Differentially Sensitizes Malignant Glioma Cells to TRAIL/Apo2L-Mediated Apoptis Through Activation of Prospases and Release of Cytochrome c from Mitichondria," Journal of Experimental Therapeutics & Oncology 5, no. 1 (2005): 39-48.
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